On Gays & Lesbians

02-26-18

Transgender Ideology Is Funded by hundreds of Millions from Drug, Surgery and Healthcare Investors

Research shows that wealthy transgender donors and medical investors who stand to benefit financially from sex-change treatments, have spent more than $400 million to promote the claim that men and women should be free to switch their 'gender' whenever they want to do so. The ideology also demands that parents and doctors, reporters, researchers, and surgeons stay silent in the face of growing evidence that the ideology and treatments are hazardous to the subjects' physical health and mental stability. Most of the donated money goes to normalizing the transgenderism

Full Article

09-23-22 Motive Behind Vanderbilt Pediatric Transgender Clinic Exposed as a ‘Big Money Maker’

Journalist Matt Walsh reported that Vanderbilt University in Tennessee got into the gender transition game, which includes hormone drugs and transition surgeries for minor children that permanently disfigure their bodies and cause sterility, in large part because it is very financially profitable. University authorities then threatened any staff members who objected, and enlisted a gang of trans activists to act as surveillance in order to force compliance.

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Vanderbilt University Medical Center (VUMC) has deleted the website of their Transgender Clinic after journalist Matt Walsh detailed a doctor’s promotion of transgender surgeries as a “big money maker” for the institution.

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Videos obtained by Walsh also show apparent threats made against VUMC medical professionals who objected to the procedures, even for religious reasons.

File called earlydioxin.htm appended here:

Early Dioxin Exposure Hinders Sperm Later

An explosion at a chemical factory near Seveso, Italy, in 1976 exposed factory workers and local residents to the pollutant dioxin, presenting an opportunity to track how exposure at different ages affects sperm quality.

Now, results from a new study point to a window of vulnerability in reproductive system development when such chemical disruptions may leave a permanent mark. "The timing is very important," says study co-author Larry Needham of the Centers for Disease Control and Prevention (CDC) in Atlanta.

Led by Paolo Mocarelli of the University Milano–Bicocca in Milan, Italy, researchers analyzed levels of dioxin in blood samples taken in 1976 from 135 exposed males. They collected new samples of blood and sperm from these same men, and compared those data to samples from a control group of 184 men.

For complete article and references see "Science News Week of Feb. 9, 2008; Vol. 173, No. 6 , p. 94."

File called drugs in waterways appended here:

Drugs in Waterways

States Worry about Drugs in Waterways

The Washington Post 6-24-2005

Washington – Academies, state officials and environmental advocates are starting to question whether massive amounts of [Forbidden WEB site? "http://www.pacifier.com/%7Eaxis/S23Drugs.html] discarded pharmaceuticals, which are often flushed down the drain, pose a threat to the nations aquatic life and possibly to people.

In waterways from the Potomac to the Brazos River In Texas, researchers have found fish laden with [Link Missing]estrogen and antidepressants and many show evidence of major neurological or physiological changes.

No one has seen evidence of effects on human health, but a number are asking publicly why the federal government is not taking a more aggressive approach to what they see as a looming problem.

In October 2002, Main's Department of Environmental Protection asked federal scientists to analyze water samples to determine to what extent prescription drugs had seeped into the states waterways. Worried that discarded birth-control pills, antidepressants and other drugs could affect the states fishing industry and public health, the departments Ann Pistell hoped the federal Environmental Protection Agency's Northwest regional office could give her a speedy answer.

It was 2 & 1/2 years before she received a partial report identifying drugs in the water without a detailed explanation, – it came in the past week – she said she is still waiting for a full breakdown.

“We're sort of baffled and frustrated by the lack of a sample analysis,” said Pistell, an environmental specialist. “We see this as an emerging issue. The more we find out, the more concerned we are.”

File called "cancercausinghormone" appended here:

Cancer Causing & Hormone Disrupting Chemicals Found in Most U.S.Homes

* * * ENDOCRINE DISRUPTERS FOUND EVERYWHERE IN HOMES * * *

Kellyn Betts, Environmental Science & Technology, Sept. 22, 2003

The air and dust inside U.S. homes are likely to contain a wide variety of chemicals and pesticides that have been identified as endocrine-disrupting compounds, according to research posted to ES&T's Research ASAP website this week.The most comprehensive analysis conducted to date, it reveals that many people may be continually exposed to dangerous levels of toxic substances, including chemicals like DDT and PCBs, which have been banned for decades.

continued @ http://www.organicconsumers.org/foodsafety/endocrine100903.cfm

• Hormone-Disrupting Chemicals. Vol. 23, ts, 11-01-1998, pp 1-4.
• HDC, The "Effects" and how it "Affects"
• Enviromental Health Perspectives;
• Organic Consumers Union; "Cancer Causing & Hormone Disrupting Chemicals" Found in Most U.S.Homes.
• Our Stolen Future, The book Our Stolen Future brought world-wide attention to scientific discoveries about endocrine disruption and the fact that common contaminants can interfere with the natural signals controlling development of the fetus. This [Link Missing] website tracks the most recent developments.
• Hormone-Disrupting Chemicals in San Francisco Bay
• Chemicals, products, and regulatory failure: A prescription for greener chemistry and better public health
• EPA recognizes that organochlorine pesticides may cause endocrine disruption
• Natural Resources Defense Council Endrocin Disrupters

*************

File called "diabetesfromplastic" appended here. Diabetes from a Plastic?

Estrogen mimic provokes insulin resistance

Exposure to small amounts of an ingredient in polycarbonate plastic may increase a person's risk of diabetes, according to a new study in mice.

The synthetic chemical called bisphenol-A is used to make dental sealants, sturdy microwavable plastics, linings for metal food-and-beverage containers, baby bottles, and numerous other products. When consumed, the chemical can mimic the effects of estrogen. Previous tests had found that bisphenol-A can leach into food and water and that it's widely prevalent in human blood. · · ·

http://www.sciencenews.org/articles/20060121/fob4.asp

Adverse Health Effects of Plastics

In addition to creating safety problems during production, many chemical additives that give plastic products desirable performance properties also have negative environmental and human health effects. These effects include:

• Direct toxicity, as in the cases of lead, cadmium, and mercury
• Carcinogens, as in the case of diethylhexyl phthalate (DEHP)
• Endocrine disruption, which can lead to cancers, birth defects, immune system suppression and developmental problems in children.

Chemical Migration from [Link Missing] Plastic Packaging into Contents

People are exposed to these chemicals not only during manufacturing, but also by using plastic packages, because some chemicals migrate from the plastic packaging to the foods they contain. examples of plastics contaminating food have been reported with most plastic types, including Styrene from polystyrene, plasticizers from PVC, antioxidants from polyethylene, and Acetaldehyde from PET.

12-07 On the news recently one quick little blurb about DEHP in baby bottles.

File called "perchlorate" appended here.

Perchlorate pollutant masculinizes fish

They proved to be hermaphroditic females

Science News
August 12, 2006 Vol 170 pg 99

By: Janet Raloff

Known largely as a component of rocket fuel, perchlorate is a pollutant that often turns up in soil and water. In dozens of studies, it has perturbed thyroid-hormone concentrations, which can affect growth and neurological development. Data from fish now indicate that perchlorate can also disrupt sexual development. Some of the changes were so dramatic that scientists initially mistook female fish for males. Several females displayed male-courtship behavior and produced sperm.

Richard R. Bernhardt of the University of Alaska in Anchorage and his colleagues focused on threespine sticklebacks (Gasterosteus aculeatus), a tiny marine species. For 3 weeks, the researchers incubated wild-captured adults in clean water or in water treated with 30, 60, or 100 parts per million (ppm) perchlorate. The adults spawned during that period.

Each group's offspring were then raised to sexual maturity in similarly treated or untreated water. At spawning age, 10 apparent males per treatment group were each given their own aquariums. Once a day, each male received a 10-minute v isit from an egg-swollen female in the same treatment group.

The first sign of something amiss: Among perchlorate-exposed fish, many would-be dads lacked the electric-blue and red coloration that normally signals readiness to spawn. Most of these fish didn't exhibit typical reproductive behaviors, such as nest building, and many ignored prospective mates. Among clean-water males, 80 percent spawned. As the perchlorate concentration climbed from 30 to 100 ppm, successful spawning fell from 50 percent to zero.

Eventually, the bellies of three apparent males began swelling with eggs. They proved to be hermaphroditic females, bearing both fertile eggs and fertile sperm.

The perchlorate-exposed true males developed unusually long testes.

Last January, the Environmental Protection Agency suggested limiting perchlorate contamination in natural bodies of water to 24.5 parts per billion. The concentrations used in the new study were at least 1,000 times that limit.

However, these doses are still environmentally relevant, argue aquatic toxicologist Bernhardt and his colleagues in the August Environmental Toxicology and Chemistry. They say that the test concentrations are similar to or less than those at several contaminated U.S. sites.

The "big surprise" was that perchlorate could produce hermaphroditic females and males with superlarge testes, says ecotoxicologist Gerald T. Ankley of EPA's Mid-Continent Ecology Division in Duluth, Minn. Clearly, that's "not something you would have anticipated [from] the way we think perchlorate works," he adds.

All the changes observed suggest that perchlorate "is acting like an androgen," or male-sex hormone, notes fish physiologist Ann Cheek of the University of Texas Health Science Center at Houston. Confirming this would require cellular analyses of testes and thyroid tissue.

Christopher W. Theodorakis of Southern Illinois University in Edwardsville argues that the "intriguing" masculinization may instead point to a new role for thyroid hormones—preservation of reproductive function.

"This paper may be telling us there's more to perchlorate—and its effects on the thyroid—than we'd realized," agrees R. Thomas Zoeller, a thyroid endocrinologist at the University of Massachusetts in Amherst. That "could be pretty profound," he says.

File called "perchloratepump" appended here.

Perchlorate Pump: Molecule draws contaminant into breast milk

Sarah C. Williams

A molecular pump designed to transport iodine also concentrates the pollutant perchlorate in breast milk, scientists have shown. The result is higher levels of the chemical in breast milk than in other parts of the body, with implications for the amount of perchlorate that pregnant and lactating women can safely ingest.

For the past decade, scientists have debated the health effects of perchlorate, which leaches into groundwater around explosives manufacturing plants. The contaminant was found in some states' drinking water in the mid-1990s. Since then, perchlorate has also turned up in produce grown with contaminated water, and scientists and policy makers have reached no consensus on what levels of the chemical are safe in humans.

Scientists knew that iodine deficiency can cause developmental problems for a nursing newborn. The new results show how perchlorate blocks iodine's movement into organs like the thyroid, lactating breast, salivary glands, and stomach by interfering with a molecular pumping system known as the Na+/I- symporter(NIS). NIS normally raises iodine levels in those organs to more than 40 times the concentrations in the bloodstream.

Ordinarily NIS occupies a cell membrane and simultaneously transports two positively charged sodium ions and one negatively charged iodine, establishing a charge difference across the membrane. But when Nancy Carrasco of the Albert Einstein College of Medicine in New York City and her colleagues exposed cells with NIS to both perchlorate and iodine, no charge difference developed, suggesting that the perchlorate blocked NIS from pumping any ions at all.

But the NIS, it turns out, was pumping the perchlorate instead of the iodine, the team reports online and in an upcoming Proceedings of the National Academy of Sciences.

When the researchers arranged cells with NIS on a filter between two compartments, and put iodine and sodium in the contraption, NIS moved the ions across the filter, concentrating iodine on one side.

When they added perchlorate, though, the iodine stayed put. For about an hour, NIS pumped only perchlorate across the filter. It was only after all the perchlorate had been removed that NIS started pumping iodine again.

"NIS is actually moving perchlorate first, but once that's depleted it still can move the iodine," says Carrasco. "This confirms that NIS has a higher affinity for perchlorate than iodine."

For a lactating mother, this means that perchlorate does double damage, making breast milk not only iodine deficient but also full of perchlorate, which further depletes the baby of iodine.

To explain why their first results, based on charge, had been misleading, the researchers watched radioactively labeled versions of a molecule similar to perchlorate interact with NIS. They realized that when NIS transports a negatively charged perchlorate ion across a membrane, only one positive sodium accompanies it—instead of the two that NIS transports through with iodine.

Purnendu Dasgupta of the University of Texas at Arlington, who in 2005 was the first to note the presence of perchlorate in breastmilk, is not surprised by the results.

"The relatively large concentrations of perchlorate in mothers' milk have suggested, even before this study, that perchlorate is actively transported," he says. "This is the first study that unequivocally shows it."

Dasgupta says this molecular proof heightens his concern that pregnant women need more iodine than most currently get, arguing that this would combat the nearly unavoidable levels of perchlorate that people unknowingly ingest.

"If in this country, people would spend half as much time worrying about iodine nutrition than about perchlorate, we would be much better off," he says.

If you have a comment on this article that you would like considered for publication in Science News, send it to editors@sciencenews.org. Please include your name and location.

References:

Dohán, O. . . . and N. Carrasco. In press. Na+/I– symporter (NIS) mediates electroneutral active transport of the environmental pollutant perchlorate. Proceedings of the National Academy of Sciences. Abstract available at http://dx.doi.org/10.1073/pnas.0707207104.

Sources:

• Nancy Carrasco
• Department of Molecular Pharmacology
• Albert Einstein College of Medicine
• Bronx, NY 10461
• Purnendu K. (Sandy) Dasgupta
• Department of Chemistry and Biochemistry
• University of Texas, Arlington
• Arlington, TX 76019-0065

File called "fetal-egg development" appended here.

Science News
Feb 3, 2007
vol 171 #5 pg 78

Plastics ingredient disrupts fetal-egg development

A common estrogen-mimicking chemical can damage eggs while an animal is still in the womb, researchers report.

Bisphenol A is found in polycarbonate plastics— those used to make baby bottles and hard-shell water bottles— and in the lining of food cans. The chemical also turns up in human tissues at concentrations of several parts per billion.

Earlier research had linked bisphenol A to reproductive problems in male and female mice. In 2003, molecular geneticist Patricia A. Hunt of Washington State University in Pullman and her colleagues exposed female mice to doses of the chemical typical of environmental concentrations. This increased the likelihood that eggs would have abnormal numbers of chromosomes (SN: 4/5/03, p. 213: Available to subscribers at http://www.sciencenews.org/articles/20030405/fob6.asp).

But "the process of making an egg is incredibly long," notes Hunt. Egg development begins in the female fetus, stops before birth, and then resumes just before ovulation. To look for effects of exposure during the earlier developmental phase, Hunt's team implanted bisphenol A pellets in pregnant mice. The pellets released the same dose used in the group's earlier experiment.

The researchers compared eggs from the female offspring of these pregnant mice with eggs from mice whose mothers had carried a placebo pellet. Up to 40 percent of the eggs from females exposed to bisphenol A as fetuses had abnormal numbers of chromosomes, the group reports in the January PLoS Genetics. Only about 3 percent of the placebo group showed that abnormality.

"The mother's exposure is influencing the genetic quality of her grandchildren," says Hunt.

File called 'Gender Measure' appended here:

Gender Measure: Pollutant appears to alter boys' genitals

Common plasticizers and solvents developed genital changes

Science News Magazine, June 4, 2005.    Vol 167  #23
by Janet Raloff and Ben Harder

Infant boys who were exposed in the womb to modest concentrations of certain common plasticizers and solvents developed genital changes including smaller-than-normal penises, a new study finds.

The results from this study of 85 boys are consistent with what researchers have seen in laboratory animals treated with the chemicals, which are called phthalates. In such tests, the substances impair fetal production of testosterone and other male sex hormones (SN: 4/3/99, p. 213: http://www.sciencenews.org/pages/sn_arc99/4_3_99/fob3.htm). http://www.sciencenews.org/articles/20050604/a6214_1550.jpg

In the current study, mothers with the highest phthalate exposures bore boys with slightly less space between the gonads and anus than did mothers with less phthalate exposure, as gauged by the women's urine concentrations of the chemicals during pregnancy. Moreover, boys with a short anogenital distance tended to have smaller penises and were far more likely to have testes that didn't descend properly into the scrotum.

Anogenital distance is typically longer in males than in females. In rodents, prenatal phthalate exposure can erase this difference, says Paul M.D. Foster of the National Institute of Environmental Health Sciences in Research Triangle Park, N.C. He's shown that this shortening in male animals can signal major permanent impairments in reproductive organs (SN: 9/2/00, p. 152: http://www.sciencenews.org/articles/20000902/bob9.asp.

In people, the anogenital distance "is not a measurement that is typically made," notes J. Bruce Redmon, an endocrinologist at the University of Minnesota Medical School in Minneapolis and an investigator in the new study.

In their research, he and his colleagues linked the signs of demasculinization to far lower phthalate exposures than have previous studies involving animals, notes study leader Shanna Swan of the University of Rochester (N.Y.) School of Medicine and Dentistry. Other research has already shown that more than one-quarter of U.S. women have phthalate concentrations in their bodies greater than those deemed in the new study to have genital-altering effects.

The data from boys suggest that in people, phthalates "may reduce the production of testosterone by the fetal testis, with subsequent downstream consequences," says Richard M. Sharpe of the University of Edinburgh's Centre for Reproductive Biology in Scotland. Such effects include low sperm production in adulthood and increased risk of testicular cancer, he says.

For the new report, Swan and her colleagues regularly examined pregnant women in several cities until the births of their babies. The researchers made a battery of measurements on the babies shortly after birth (SN: 11/13/04, p. 318: Available to subscribers at http://www.sciencenews.org/articles/20041113/note13.asp.

The anogenital distances for boys exposed to the highest amounts of phthalates were 19 percent shorter, on average, than those of boys less exposed. Although that's not a large enough difference for most doctors to notice, Sharpe says, the finding is "potentially, a big deal."

The study results, which will appear in an upcoming Environmental Health Perspectives, show a statistically strong association between a short anogenital distance and high exposures to four phthalates. These chemicals were diethyl phthalate, used in perfumes; dibutyl phthalate, used in nail polish and hard plastics; butyl benzyl phthalate, used primarily in flooring; and diisobutyl phthalate, used in paints, adhesives, and polyvinyl chloride plastics.

Referring to the new study, Marian K. Stanley of the chemical industry's Phthalate Esters Panel observes, "It's hard to know what to make of it." She says that there's no definition of a normal anogenital distance in babies. Stanley also observes that diethyl phthalate, which the new study links to shortened anogenital distances, has no such effect in lab animals.

Although Redmon agrees that the norm for boys is unknown, he adds that a reduced anogenital distance "is what you would expect" if phthalates lower the concentrations of male hormones in the human fetus.

Regarding diethyl phthalate, Swan notes that several studies have linked the chemical to reproductive effects in people.

Stanley, Redmon, and others agree that the infants in the current study should be followed for several years. They also call for studies based on similar measurements in larger groups of pregnant women and their infants.

File called 'ignore_estrogen_in_soy.htm' appended here:

Estrogen in Soy

Science News Volume 155, Number 20 (May 15, 1999)

References & Sources

The ignored estrogen in soy

Of the three estrogen-mimicking compounds in soy, glycitein occurs in the smallest quantities, but it appears the most potent and readily absorbed.

References:

Song, T.T., S. Hendrich, and P.A. Murphy. 1999. Estrogenic activity of glycitein, a soy isoflavone. Journal of Agricultural and Food Chemistry 47(April):1607.

Zhang, Y. . . . P.A. Murphy, et al. 1999. Urinary disposition of the soybeanisoflavones daidzein, genistein and glycitein differs among humans with moderate fecal isoflavone degradation activity. Journal of Nutrition 129(May):957.

Further Readings:

Helmuth, L. 1999. Nutritionists debate soy's health benefits. Science News155(April 24):262

Raloff, J. 1999. Soy compounds help preserve bone. Science News 155(Jan.2):15.

______. 1998. Soya-nara, heart disease. Science News 155(May 30):348.

______. 1997. Plant estrogens may ward off breast cancer. Science News 152(Oct. 11):230.

______. 1997. Radical prostates. Science News 151(Feb. 22):126.

______. 1995. Eating soy to lower cholesterol. Science News 148(Aug. 19):127.

Sources:

Patricia A. Murphy

Iowa State University, Food Science and Human Nutrition

2312 Food Sciences Building

Ames, IA 50011

From Science News, Vol. 155, No. 20, May 15, 1999, p. 319. Copyright © 1999, Science Service.

File called 'environmental_hormones.htm' appended here:

Our Pollutants could be changing our sex

PBS

Environmental Hormones in the News

News stories on environmental threats from hormone mimics abound these days. Many focus on the potential breast-cancer risks posed by a woman's exposure to pollutants that function like estrogen.

Then there are the reproductive scares. In certain [Link Missing]wildlife populations, females appear to have lost the ability to successfully reproduce. In others, male animals have been born with feminized reproductive organs. There are even provocative, though spotty, human data suggesting that males may be suffering from unusual rates of genital malformations and testicular cancer, and from unusually low rates of sperm production.

Perhaps most troubling, the proportion of male births appears to be waning in many industrial countries. The source of the pollutants suspected of triggering at least some of these dire effects include common pesticides, plasticizers, fuel additives, and surfactants. Not only do trace quantities of them lace our air and drinking water, but some have been detected leaching into our foods--from plastic lined cans, from plastic coated papers, and from plastic bottles.

Many animals, including various types of prized game fish, including lake trout and salmon, build up significant stores of these compounds in their edible flesh. Some of these chemicals even appear to leach directly into saliva from teeth treated with popular dental sealants.

Though the story of hormone-mimicking pollutants has been brewing for decades, even as recently as 5 years ago, only a handful of reporters had begun looking into it. The vast majority of their brethren wouldn't follow suit for another 3 years--until the publication of "Our Stolen Future," a book by zoologist Theo Colborn, biologist John Peterson Myers, and then-Boston Globe reporter Dianne Dumanoski. Since 1994, Colborn has been on a soapbox arguing that a series of disturbing and related trends suggest an environmental pandemic may be in the making--some subtle and malign productive impairment throughout the length and breadth of Earth's animal kingdom.

Initially, she ascended that soapbox only in the supportive company of fellow biologists. Gradually, Colborn became more outspoken about her fears, catching the attention of Congress, U.S. regulatory agencies, and captains of industry. Then, with "Our Stolen Future"--and the news blitz it evoked throughout respected major media leaders--she and her co-authors carried her chilling concerns to the general public, much as Paul Brodeur's" Zapping of America" brought the formerly arcane subject of electromagnetic fields to public attention.

Our "Stolen Future" offered solid background on the secret life of our bodies' endocrine system--and made for surprisingly good drama. Anecdotes and personality profiles humanized the science of our environment's hormonal threats to wildlife and its stewards. But even when the book debuted in March 1996, most of the events it portrayed were quite dated. Moreover, much of the research that it described may ultimately prove misleading. Indeed, that's the nature of science.

Even today, research on endocrine disrupter's remains in its infancy. As a result, the trends portrayed by news articles must necessarily be sketched from a remarkably small number of data points. Such a paucity of data leaves reporters or book authors free to extrapolate broadly on what the few, early findings suggest. As a field develops, however, it traditionally constrains the boundaries of what is real--or realistic.

So where today we are questioning to what extent estrogen-mimicking pollutants may reduce sperm counts in men, we might one day learn that only three of the dozens of "man-made" estrogens to which we are exposed employ a mechanism that can affect human sperm production. Or we might find out that any of these agents can affect sperm, but only if a man's mother had been exposed to them on day 17 of her pregnancy. Or only if the fatally exposed man smoked during his teen years. Or faced exposure to these "hormones" through the skin. Or... You get the point; the probability of any serious risks may be highly circumscribed. Which is why it's exciting to write about these issues today--while they're still so deliciously appalling, and nobody has proven that even the most outlandish prospects are categorically impossible.

A few individuals have begun suggesting that they might be, however. Gina Kolata in a March 1996 New York Times piece, for instance, quoted scientists who argued that concerns about hormone mimics must be considered premature until follow-up data demonstrate not only how pervasive and potent such agents are, but sciJanalso whether the initial studies were conducted appropriately. About the same time, a related story in the Washington Post by Rick Weiss and Gary Lee picked up the skepticism, noting that science "is better at scaring people than reassuring them."

Indeed, they charged that the mix of science, politics and public relations associated with Colburn's public campaign to put the issue of "endocrine disrupter's" firmly in the public eye "smells undeniably of spin but is nevertheless difficult to ignore." Anxious to contribute to that spin have been several chemical industry groups that have begun funding a few scientists to challenge the concerns that Colborn champions. To date these critics have largely pointed to those gaps in the data supporting the frightening trends and to a host of potentially countervailing effects of chemicals in the environment--ones that might ultimately block hormone action.

The Competitive Enterprise Institute, based in Washington D.C., took up the latter crusade in a report it issued at a press conference two days before the release of Colborn's book. It noted that Mother Nature has imbued some 173 plants (at last count) with hormone mimicking constituents--many of which we have been eating without harm for millennial. As such, it argued, because the hormonal alter ego of many pesticides and other synthetic chemicals is nothing new or unusual, Colborn's campaign to even infer otherwise represents "scare tactics" that "hit below the belt." Data gaps, and the hazards of offering speculations to bridge them, plague every fledgling field of science. But the initial trends that sparse data suggest can be bolstered or responsibly discredited only by additional research.

File called 'pollutantsss.htm' appended here:

Pollutant Waits to Smite Salmon at Sea

http://www.sciencenews.org/sn_arc99/5_8_99/content.htm

Science News Magazine Vol 155 #19 May 8 1999

Canadian scientists have identified the likely culprit behind some historic, regional declines in Atlantic salmon. The researchers find that a near-ubiquitous water pollutant can render young, migrating fish unable to survive a life at sea.

Heavy, late-spring spraying of forests with a pesticide laced with nonylphenol during the 1970s and '80s was the clue that led the biologists to unmask that chemical's role in the transitory decline of salmon in East Canada. Though these sprays have ended, concentrations of nonylphenols in forest runoff then were comparable to those in the effluent of some pulp mills, industrial facilities, and sewage-treatment plants today. Downstream of such areas, the scientists argue, salmon and other migratory fish may still be at risk.

Nonylphenols are surfactants used in products from pesticides to dishwashing detergents, cosmetics, plastics, and spermicides. Because waste-treatment plants don't remove nonylphenols well, these chemicals can build up in downstream waters (SN: 1/8/94, p. 24).

When British studies linked ambient nonylphenol pollution to reproductive problems in fish (SN: 2/26/94, p. 142), Wayne L. Fairchild of Canada's Department of Fisheries and Oceans in Moncton, New Brunswick, became concerned. He recalled that an insecticide used on local forests for more than a decade had contained large amounts of nonylphenols. They helped aminocarb, the oily active ingredient in Matacil 1.8D, dissolve in water for easier spraying.

Runoff of the pesticide during rains loaded the spawning and nursery waters of Atlantic salmon with nonylphenols. Moreover, this aerial spraying had tended to coincide with the final stages of smoltification--the fish's transformation for life at sea.

To probe for effects of forest spraying, Fairchild and his colleagues surveyed more than a decade of river-by-river data on fish. They overlaid these numbers with archival data on local aerial spraying with Matacil 1.8D or either of two nonylphenol-free pesticides. One contained the same active ingredient, aminocarb, as Matacil 1.8D does.

Most of the lowest adult salmon counts between 1973 and 1990 occurred in rivers where smolts would earlier have encountered runoff of Matacil 1.8D, Fairchild's group found. In 9 of 19 cases of Matacil 1.8D spraying for which they had good data, salmon returns were lower than they were within the 5 years earlier and 5 years later, they report in the May Environmental Health Perspectives. No population declines were associated with the other two pesticides.

The researchers have now exposed smolts in the laboratory to various nonylphenol concentrations, including some typical of Canadian rivers during the 1970s. The fish remained healthy--until they entered salt water, at which point they exhibited a failure-to-thrive syndrome.

"They looked like they were starving," Fairchild told Science News. Within 2 months, he notes, 20 to 30 percent died. Untreated smolts adjusted normally to salt water and fattened up.  Steffen S. Madsen, a fish ecophysiologist at Odense University in Denmark, is not surprised, based on his own experiments.

To move from fresh water to the sea, a fish must undergo major hormonal changes that adapt it for pumping out excess salt. A female preparing to spawn in fresh water must undergo the opposite change. Since estrogen triggers her adaptation, Madsen and a colleague decided to test how smolts would respond to estrogen or nonylphenol, an estrogen mimic. In the lab, they periodically injected salmon smolts with estrogen or nonylphenol over 30 days, and at various points placed them in seawater for 24 hours. Salt in the fish's blood skyrocketed during the day-long trials, unlike salt in untreated smolts. "Our preliminary evidence indicates that natural and environ- mental estrogens screw up the pituitary," Madsen says. The gland responds by making prolactin, a hormone that drives freshwater adaptation.

Judging by Fairchild's data, Madsen now suspects that any fish that migrates between fresh and salt water may be similarly vulnerable to high concentrations of pollutants that mimic estrogen.

References:

Brown, S.B. . . . and W.L. Fairchild. 1998. Effects of water-borne 4-nonylphenol on Atlantic salmon (Salmo salar) smolts. SETAC 19th Annual Meeting. November.

Charlotte, N.C.

Fairchild, W.L., et al. 1999. Does an association between pesticide use and subsequent declines in catch of Atlantic salmon (Salmo salar) represent a case of  endocrine disruption? Environmental Health Perspectives 107(May):349.

File called 'estrogen_minnows.htm' appended here:

Estrogen Effects Linger in Male Fish

Estrogen-mimicking pollutants

Science News 8/5/00 p.94

Estrogen-mimicking pollutants can trigger gender-bending effects in wildlife. For instance, male fish exposed to such hormonally active pollutants will make vitellogenin an egg-yolk protein that's normally fashioned only by females (SN: 1/8/94, p. 24). A new study finds that once initially spurred to make vitellogenin, males don't need a steady bath of estrogen to maintain high levels of the motherly protein.

Grace H. Panter of AstraZaneca in Brixham, England, and her colleagues exposed male fathead minnows to up to 120 parts per trillion (ppt) estrogen in water for 3 or 6 weeks. Some fish lived in tanks continuously laced with the hormone. Other fish spent half their time in clean water, either every other day or 3 days straight of every 6.

Minnows intermittently exposed to 120 ppt estrogen produced almost as much vitellogenin as those continuously bathed in that amount, Painter's team found. These fish also had roughly seven times as much vitellogenin as did males that had been continuously exposed to 60 ppt-the average amount the intermittent group experienced.

Most unexpectedly, even long interludes in clean water didn't substantially erase the hormone's impact. Male minnows spending 3 weeks in 120 ppt estrogen, followed by an equal time in clean water, ended up with vitellogenin concentrations " not significantly different" from those of fish that had spent the entire 6 weeks in the estrogen-tainted water, note Panter and her coworkers.

They report their findings in the July 1 Environmental Science & Technology.

No one yet knows what the long-term consequences are for male fish that have made vitellogenin. However, members of Panter's team previously showed that exposure to estrogenic pollution during a male fish's development can stunt the growth of its testes (SN: 2/26/94 p.142).

www.dumbellnebula.com/soy.htm
http://website.lineone.net/~mwarhurst/
http://www.epa.gov/scipoly/oscpendo/
http://www.tmc.tulane.edu/ecme/eehome/
http://womenshealth.about.com/health/womenshealth/library/weekly/aa061599.htm

Neonatal intensive care units routinely save the lives of extremely premature and critically ill newborns. Many of these successes are made possible by tubing and other equipment rendered flexible with a plasticizer known as DIETHYLHEXYL PHTHALATE (DEPH). A new study finds that this equipment releases DEHP into babies, though the impact on such children is still uncertain. Do you want to be first to find out?

Plastic baby "www dot cbsnews dot com/htdocs/pdf/BabysToxicBottle.pdf missing" milk bottles may contain and leach bisphenol 'b'. Update, since this was written the dangers of bisphenol have been exposed and most manufactures now say they don't use it.

*** Damned Important ***

KEY INFORMATION

We are still talking about homosexuality

Science News 11-02-02 Vol 162 # 18 an article, "More Frog Trouble, Herbicides may emasculate wild males." We are talking Atrazine here and in extremely small concentrations on the order of 0.5 ppb. You don't think your affected? Atrazine can be found in practically all rain water. (4-15-02) It was on CNN that Atrazine (primary component of Round-Up ® etc.) found in extremely low levels was causing newborn frogs (tadpoles) to have both or no sex organs. But that only happens to frogs, right? And from the ENN site. "We  have so completely hobbled the EPA in this process that there is not much room for hope for pesticide control," said Mike Casey, an EWG spokesman.

Hormone Disrupting Chemicals

Our bodies carry plastic-bpa residues of kitchen plastics.

HDC, in your milk bottle could be changing your child's sexuality.

[HDC] could easily be one of the reason for much of today's sexual 'problems'.

You really need to know what they are and what they are doing to you, your family and your neighbors. British men are less fertile than hamsters. The problem is it isn't just the British. The media in the U.S. couldn't publish the info, for fear of retribution in our courts. That and the fact their biggest client is the pharmaceutical industry. ($)

Isn't it ironic, the very corporations they are protecting are turning their children into infertile queers?

More - - - frog news. But, of course; humans are unaffected <sic>.

So, if the genes are damaged, doesn't it stand to reason that it is going to affect what you turn into?

Beauty products may damage fetal development as reported in Science News Magazine.

I was thinking about the Politicians and the way they will say or do anything to stay in power. The government/industry is using the Gay controversy, just as they used the UFO hysteria to hide military secrets, they are using it to hide their non response to industrial pollution. It confounds me that this never hit the media or was never under investigation, but; then again, it makes sense.

The government/industry doesn't want you to make the connection between pollution and gene abnormalities because they are getting millions of dollars from those very people to hide that fact.

The effects of combinations of low level wastes (we are talking parts per billion) that combined; multiplied their effects on the body which effectively put the government toxic standards, which tests only one substance at a time; almost pointless, ludicrous at best.

Another story (drugs in the waterways) that points out the problem. There is something in the news every few weeks related to this, if you only take the time to look.

Paper mill pollutants contain a time bomb.

The 20 year old article that proves nothing is going to change unless we do something.

Do you think being homosexual is a choice? It is about as much choice as being born autistic, blind, or deaf. I think the GAY issue is the second biggest hoax ever pulled on Americans of all time and the reason for it is the same. What if the [link missing] food industry was selling the Americans something that affected them in such a way their genes were screwed up. Do you think that they would do it? Don't tell me you don't think one American (or anybody else for that matter) would screw another American just for the sake of money? [sic]

How could the GAY thing be a [ link disconnected "truthaboutsoy.htm"] soy conspiracy? The evidence is overwhelming but I think the media along with government & government officials are being bribed and threatened and are so petrified of losing the liberal vote and of course it is easier to go along with them then opening this gigantic can of ugly worms.

The gay issue seems to be something everybody is pussy footing around feeling there is no answer. Or, call me paranoid; but the government would rather it be called an individuals choice rather than the real answer. It takes the heat off them, at least until the election is over.

December 2006, in the news. When women quit taking "hormone replacements" due to recommendation only a few months ago, there was a drastic reduction in breast cancer, like 15% in only a few months. That is huge! So, how many things have I been saying has estrogen or estrogen mimicking chemicals in it?

I think it has to do with our environment and the blatant poisoning of our populace. It has to do with industry and who is paying who off. Did it occur to you as being odd that the 'plastics' industry is putting big bucks into advertising (pumping up their image) for no apparent reason? Bet me! They never spend big bucks without a reason.  There are innumerable examples on how things like this affect us but nobody seems to want to put it together.

I think it measures up to a full blown conspiracy.

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Militant homosexual groups “Lobbied” to get Homosexuality removed from the List of “Mental Illness” ailments